Identification of N-arachidonylglycine as the endogenous ligand for orphan G-protein coupled receptor GPR18. (abst – 2006) http://www.ncbi.nlm.nih.gov/pubmed/16844083/
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2865488/
GPR18 in microglia: implications for the CNS and endocannabinoid system signaling (full – 2012) http://onlinelibrary.wiley.com/doi/1...2.02019.x/full
siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycine-induced cell migration (full – 2012) http://www.jmolecularsignaling.com/content/7/1/10
So what do we call GPR18 now? (full – 2012) http://onlinelibrary.wiley.com/doi/1...1.01731.x/full
Δ9-Tetrahydrocannabinol and N-arachidonyl glycine are full agonists at GPR18 receptors and induce migration in human endometrial HEC-1B cells (full – 2012) http://onlinelibrary.wiley.com/doi/1...1.01497.x/full
N-arachidonoyl glycine induces macrophage apoptosis via GPR18. (abst – 2012) http://www.ncbi.nlm.nih.gov/pubmed/22266325
The Concise Guide to PHARMACOLOGY 2013/14: G Protein-Coupled Receptors (full – 2013) http://onlinelibrary.wiley.com/doi/1...bph.12445/full
Do N-arachidonyl-glycine (NA-glycine) and 2-arachidonoyl glycerol (2-AG) share mode of action and the binding site on the β2 subunit of GABAA receptors? (full – 2013) https://peerj.com/articles/149/
N-Arachidonyl glycine does not activate G protein-coupled receptor 18 signaling via canonical pathways. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3533477/
N-arachidonoyl-L-serine (AraS) possesses proneurogenic properties in vitro and in vivo after traumatic brain injury. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3734775/
The Natural Product Magnolol as a Lead Structure for the Development of Potent Cannabinoid Receptor Agonists (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3813752/
A GPR18-based signaling system regulates IOP in murine eye. (full– 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3687663/
Mechanism of Central Atypical Cannabinoid Receptor GPR18-Mediated Hypotension in Conscious Rats (abst – 2013) http://www.fasebj.org/cgi/content/me...c-38c0c63c19ad
Role of Central Atypical Cannabinoid Receptor GPR18 in Modulating Cardiovascular Function (abst – 2013) http://www.fasebj.org/cgi/content/me...c-38c0c63c19ad
Cannabinoid receptor modulation of the endothelial cell inflammatory response (abst – 2013) http://www.jimmunol.org/cgi/content/...2-845dc670f7cf
The Novel Endocannabinoid Receptor GPR18 is Expressed in the Rostral Ventrolateral Medulla and Exerts Tonic Restraining Influence on Blood Pressure. (full – 2014) http://jpet.aspetjournals.org/conten...13.209213.long
The endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cells. (full – 2014) http://www.jbc.org/content/early/201...13.536953.long
CB2 Receptor Activation Inhibits Melanoma Cell Transmigration through the Blood- Brain Barrier. (full – 2014) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4057719/
Neuronal Nitric Oxide Synthase Dependent Elevation in Adiponectin in the Rostral Ventrolateral Medulla Underlies GPR18-mediated Hypotension in Conscious Rats. (full – 2014) http://jpet.aspetjournals.org/conten...14.216036.long
Cannabinoid and lipid-mediated vasorelaxation in retinal microvasculature. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24751709
The endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cells. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24644287
Activation of GPR18 by Cannabinoid compounds: A tale of biased agonism. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24762058
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2865488/
GPR18 in microglia: implications for the CNS and endocannabinoid system signaling (full – 2012) http://onlinelibrary.wiley.com/doi/1...2.02019.x/full
siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycine-induced cell migration (full – 2012) http://www.jmolecularsignaling.com/content/7/1/10
So what do we call GPR18 now? (full – 2012) http://onlinelibrary.wiley.com/doi/1...1.01731.x/full
Δ9-Tetrahydrocannabinol and N-arachidonyl glycine are full agonists at GPR18 receptors and induce migration in human endometrial HEC-1B cells (full – 2012) http://onlinelibrary.wiley.com/doi/1...1.01497.x/full
N-arachidonoyl glycine induces macrophage apoptosis via GPR18. (abst – 2012) http://www.ncbi.nlm.nih.gov/pubmed/22266325
The Concise Guide to PHARMACOLOGY 2013/14: G Protein-Coupled Receptors (full – 2013) http://onlinelibrary.wiley.com/doi/1...bph.12445/full
Do N-arachidonyl-glycine (NA-glycine) and 2-arachidonoyl glycerol (2-AG) share mode of action and the binding site on the β2 subunit of GABAA receptors? (full – 2013) https://peerj.com/articles/149/
N-Arachidonyl glycine does not activate G protein-coupled receptor 18 signaling via canonical pathways. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3533477/
N-arachidonoyl-L-serine (AraS) possesses proneurogenic properties in vitro and in vivo after traumatic brain injury. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3734775/
The Natural Product Magnolol as a Lead Structure for the Development of Potent Cannabinoid Receptor Agonists (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3813752/
A GPR18-based signaling system regulates IOP in murine eye. (full– 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3687663/
Mechanism of Central Atypical Cannabinoid Receptor GPR18-Mediated Hypotension in Conscious Rats (abst – 2013) http://www.fasebj.org/cgi/content/me...c-38c0c63c19ad
Role of Central Atypical Cannabinoid Receptor GPR18 in Modulating Cardiovascular Function (abst – 2013) http://www.fasebj.org/cgi/content/me...c-38c0c63c19ad
Cannabinoid receptor modulation of the endothelial cell inflammatory response (abst – 2013) http://www.jimmunol.org/cgi/content/...2-845dc670f7cf
The Novel Endocannabinoid Receptor GPR18 is Expressed in the Rostral Ventrolateral Medulla and Exerts Tonic Restraining Influence on Blood Pressure. (full – 2014) http://jpet.aspetjournals.org/conten...13.209213.long
The endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cells. (full – 2014) http://www.jbc.org/content/early/201...13.536953.long
CB2 Receptor Activation Inhibits Melanoma Cell Transmigration through the Blood- Brain Barrier. (full – 2014) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4057719/
Neuronal Nitric Oxide Synthase Dependent Elevation in Adiponectin in the Rostral Ventrolateral Medulla Underlies GPR18-mediated Hypotension in Conscious Rats. (full – 2014) http://jpet.aspetjournals.org/conten...14.216036.long
Cannabinoid and lipid-mediated vasorelaxation in retinal microvasculature. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24751709
The endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cells. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24644287
Activation of GPR18 by Cannabinoid compounds: A tale of biased agonism. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24762058