Identification of a new class of molecules, the arachidonyl amino acids, and characterization of one member that inhibits pain. (full – 2001) http://www.jbc.org/content/276/46/42639.long
Regulation of anandamide tissue levels by N-arachidonylglycine. (abst – 2002) http://www.ncbi.nlm.nih.gov/pubmed/12234618
A structure-activity relationship study on N-arachidonoyl-amino acids as possible endogenous inhibitors of fatty acid amide hydrolase (abst – 2004) http://www.ncbi.nlm.nih.gov/pubmed/14715265
N-Arachidonyl-glycine inhibits the glycine transporter, GLYT2a. (full – 2006) http://onlinelibrary.wiley.com/doi/1...6.04107.x/full
Identification of N-arachidonylglycine as the endogenous ligand for orphan G-protein coupled receptor GPR18. (abst – 2006) http://www.ncbi.nlm.nih.gov/pubmed/16844083/
Actions of N-arachidonyl-glycine in a rat inflammatory pain model. (full – 2007) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2042976/
Actions of N-arachidonyl-glycine in a rat neuropathic pain model. (abst – 2008) http://www.ncbi.nlm.nih.gov/pubmed/17588618
The endocannabinoid anandamide is a precursor for the signaling lipid N-arachidonoyl glycine by two distinct pathways (full – 2009) http://www.ncbi.nlm.nih.gov/pmc/arti...9/?tool=pubmed
N-arachidonoyl glycine (article – 2009) http://www.lipidmaps.org/update/2009...?s=cannabinoid
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2865488/
N-arachidonoyl glycine, an endogenous lipid that acts as a vasorelaxant via nitric oxide and large conductance calcium-activated potassium channels. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2931560/
Toxicology studies with N-acetylglycine. (abst – 2010) http://www.ncbi.nlm.nih.gov/pubmed/20188778
Resolution of inflammation by N-arachidonoylglycine. (full – 2011) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3196844/
siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycine-induced cell migration. (full – 2012) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3493281/
Δ9-Tetrahydrocannabinol and N-arachidonyl glycine are full agonists at GPR18 receptors and induce migration in human endometrial HEC-1B cells (full – 2012) http://onlinelibrary.wiley.com/doi/1...1.01497.x/full
N-arachidonoyl glycine induces macrophage apoptosis via GPR18. (abst – 2012) http://www.ncbi.nlm.nih.gov/pubmed/22266325
N-Arachidonyl glycine does not activate G protein-coupled receptor 18 signaling via canonical pathways. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3533477/
A GPR18-based signaling system regulates IOP in murine eye. (full– 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3687663/
The Novel Endocannabinoid Receptor GPR18 is Expressed in the Rostral Ventrolateral Medulla and Exerts Tonic Restraining Influence on Blood Pressure. (full – 2014) http://jpet.aspetjournals.org/conten...13.209213.long
Δ(9)-THC and N-arachidonoyl glycine regulate BV-2 microglial morphology and cytokine release plasticity: implications for signaling at GPR18. (full – 2014) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3877838/
Vascular targets for cannabinoids: animal and human studies. (full – 2014) http://onlinelibrary.wiley.com/doi/1...bph.12560/full
The cannabinoid acids, analogs and endogenous counterparts. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24731541
Cannabinoid and lipid-mediated vasorelaxation in retinal microvasculature. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24751709
Activation of GPR18 by Cannabinoid compounds: A tale of biased agonism. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24762058
Regulation of anandamide tissue levels by N-arachidonylglycine. (abst – 2002) http://www.ncbi.nlm.nih.gov/pubmed/12234618
A structure-activity relationship study on N-arachidonoyl-amino acids as possible endogenous inhibitors of fatty acid amide hydrolase (abst – 2004) http://www.ncbi.nlm.nih.gov/pubmed/14715265
N-Arachidonyl-glycine inhibits the glycine transporter, GLYT2a. (full – 2006) http://onlinelibrary.wiley.com/doi/1...6.04107.x/full
Identification of N-arachidonylglycine as the endogenous ligand for orphan G-protein coupled receptor GPR18. (abst – 2006) http://www.ncbi.nlm.nih.gov/pubmed/16844083/
Actions of N-arachidonyl-glycine in a rat inflammatory pain model. (full – 2007) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2042976/
Actions of N-arachidonyl-glycine in a rat neuropathic pain model. (abst – 2008) http://www.ncbi.nlm.nih.gov/pubmed/17588618
The endocannabinoid anandamide is a precursor for the signaling lipid N-arachidonoyl glycine by two distinct pathways (full – 2009) http://www.ncbi.nlm.nih.gov/pmc/arti...9/?tool=pubmed
N-arachidonoyl glycine (article – 2009) http://www.lipidmaps.org/update/2009...?s=cannabinoid
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2865488/
N-arachidonoyl glycine, an endogenous lipid that acts as a vasorelaxant via nitric oxide and large conductance calcium-activated potassium channels. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2931560/
Toxicology studies with N-acetylglycine. (abst – 2010) http://www.ncbi.nlm.nih.gov/pubmed/20188778
Resolution of inflammation by N-arachidonoylglycine. (full – 2011) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3196844/
siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycine-induced cell migration. (full – 2012) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3493281/
Δ9-Tetrahydrocannabinol and N-arachidonyl glycine are full agonists at GPR18 receptors and induce migration in human endometrial HEC-1B cells (full – 2012) http://onlinelibrary.wiley.com/doi/1...1.01497.x/full
N-arachidonoyl glycine induces macrophage apoptosis via GPR18. (abst – 2012) http://www.ncbi.nlm.nih.gov/pubmed/22266325
N-Arachidonyl glycine does not activate G protein-coupled receptor 18 signaling via canonical pathways. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3533477/
A GPR18-based signaling system regulates IOP in murine eye. (full– 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3687663/
The Novel Endocannabinoid Receptor GPR18 is Expressed in the Rostral Ventrolateral Medulla and Exerts Tonic Restraining Influence on Blood Pressure. (full – 2014) http://jpet.aspetjournals.org/conten...13.209213.long
Δ(9)-THC and N-arachidonoyl glycine regulate BV-2 microglial morphology and cytokine release plasticity: implications for signaling at GPR18. (full – 2014) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3877838/
Vascular targets for cannabinoids: animal and human studies. (full – 2014) http://onlinelibrary.wiley.com/doi/1...bph.12560/full
The cannabinoid acids, analogs and endogenous counterparts. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24731541
Cannabinoid and lipid-mediated vasorelaxation in retinal microvasculature. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24751709
Activation of GPR18 by Cannabinoid compounds: A tale of biased agonism. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/24762058