-arachidonoyl L-serine, an endocannabinoid-like brain constituent with vasodilatory properties. (full – 2006) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1413724/ N-arachidonoyl L-serine, a putative endocannabinoid, alters the activation of N-type Ca2+ channels in sympathetic neurons. (full – 2008) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2652135/
Inhibition of human neutrophil chemotaxis by endogenous cannabinoids and phytocannabinoids: evidence for a site distinct from CB1 and CB2. (full – 2008) http://molpharm.aspetjournals.org/content/73/2/441.long
Endocannabinoid-like N-arachidonoyl serine is a novel pro-angiogenic mediator. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2936832/
Angiogenesis: a new physiological role for N-arachidonoyl serine and GPR55? (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2936831/
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2865488/
N-arachidonoyl--serine is neuroprotective after traumatic brain injury by reducing apoptosis (full – 2011) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3170948/
N-arachidonoyl-L-serine (AraS) possesses proneurogenic properties in vitro and in vivo after traumatic brain injury. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3734775/
Vascular targets for cannabinoids: animal and human studies. (full – 2014) http://onlinelibrary.wiley.com/doi/1...bph.12560/full
Inhibition of human neutrophil chemotaxis by endogenous cannabinoids and phytocannabinoids: evidence for a site distinct from CB1 and CB2. (full – 2008) http://molpharm.aspetjournals.org/content/73/2/441.long
Endocannabinoid-like N-arachidonoyl serine is a novel pro-angiogenic mediator. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2936832/
Angiogenesis: a new physiological role for N-arachidonoyl serine and GPR55? (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2936831/
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor. (full – 2010) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2865488/
N-arachidonoyl--serine is neuroprotective after traumatic brain injury by reducing apoptosis (full – 2011) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3170948/
N-arachidonoyl-L-serine (AraS) possesses proneurogenic properties in vitro and in vivo after traumatic brain injury. (full – 2013) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3734775/
Vascular targets for cannabinoids: animal and human studies. (full – 2014) http://onlinelibrary.wiley.com/doi/1...bph.12560/full