Administration of 2-arachidonoylglycerol ameliorates both acute and chronic Experimental Autoimmune Encephalomyelitis (abst – 2011) http/www.unboundmedicine.com/medline/ebm/record/21406188/abstract/Administration_of_2_arachidonoylglycerol_ameliorat es_both_acute_and_chronic_Experimental_Autoimmune_ Encephalomyelitis_
Evaluation of the Effects of Sativex (THC BDS: CBD BDS) on Inhibition of Spasticity in a Chronic Relapsing Experimental Allergic Autoimmune Encephalomyelitis: A Model of Multiple Sclerosis. (full – 2012) http/www.ncbi.nlm.nih.gov/pmc/articles/PMC3423911/pdf/ISRN.NEUROLOGY2012-802649.pdf
Cannabinoid receptor-2-selective agonists improve recovery in experimental autoimmune encephalomyelitis (abst – 2012) http/www.jimmunol.org/cgi/content/meeting_abstract/188/1_MeetingAbstracts/116.7?maxtoshow=&hits=25&RESULTFORMAT=&fulltext=cannabinoid&searchid=1&FIRSTINDEX=130&sortspec=date&resourcetype=HWCIT
Cannabinoids ameliorate disease progression in a model of multiple sclerosis in mice, acting preferentially through CB(1) receptor-mediated anti-inflammatory effects. (abst - 2012) http/www.ncbi.nlm.nih.gov/pubmed/22342378
Cannabinoid receptor 2 agonists inhibit migration of activated dendritic cells via modulation of MMP-9 (abst – 2012) http/www.jimmunol.org/cgi/content/meeting_abstract/188/1_MeetingAbstracts/173.23?maxtoshow=&hits=25&RESULTFORMAT=&fulltext=cannabinoid&searchid=1&FIRSTINDEX=130&sortspec=date&resourcetype=HWCIT
Genetic Background Can Result in a Marked or Minimal Effect of Gene Knockout (GPR55 and CB2 Receptor) in Experimental Autoimmune Encephalomyelitis Models of Multiple Sclerosis. (full – 2013) http/www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0076907
Multiple sclerosis and the blood-central nervous system barrier. (full – 2013) http/www.ncbi.nlm.nih.gov/pmc/articles/PMC3562587/
Cannabidiol provides long-lasting protection against the deleterious effects of inflammation in a viral model of multiple sclerosis: A role for A2A receptors. (full – 2013) http/www.sciencedirect.com/science/article/pii/S0969996113001939
Selective CB2 receptor activation ameliorates EAE by reducing Th17 differentiation and immune cell accumulation in the CNS. (abst – 2013) http/www.ncbi.nlm.nih.gov/pubmed/24342422
Pre- and postsynaptic type-1 cannabinoid receptors control the alterations of glutamate transmission in experimental autoimmune encephalomyelitis. (abst – 2014) http/www.ncbi.nlm.nih.gov/pubmed/24440366
Activating Cannabinoid Receptor 2 Alleviates Pathogenesis of Experimental Autoimmune Encephalomyelitis Via Activation of Autophagy and Inhibiting NLRP3 Inflammasome. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/25417929
Neuroprotection in Experimental Autoimmune Encephalomyelitis and Progressive Multiple Sclerosis by Cannabis-Based Cannabinoids. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/25537576
Interaction between the protective effects of cannabidiol and palmitoylethanolamide in experimental model of multiple sclerosis in C57BL/6 mice (abst – 2015) http://www.sciencedirect.com/science...06452215000858
Evaluation of the Effects of Sativex (THC BDS: CBD BDS) on Inhibition of Spasticity in a Chronic Relapsing Experimental Allergic Autoimmune Encephalomyelitis: A Model of Multiple Sclerosis. (full – 2012) http/www.ncbi.nlm.nih.gov/pmc/articles/PMC3423911/pdf/ISRN.NEUROLOGY2012-802649.pdf
Cannabinoid receptor-2-selective agonists improve recovery in experimental autoimmune encephalomyelitis (abst – 2012) http/www.jimmunol.org/cgi/content/meeting_abstract/188/1_MeetingAbstracts/116.7?maxtoshow=&hits=25&RESULTFORMAT=&fulltext=cannabinoid&searchid=1&FIRSTINDEX=130&sortspec=date&resourcetype=HWCIT
Cannabinoids ameliorate disease progression in a model of multiple sclerosis in mice, acting preferentially through CB(1) receptor-mediated anti-inflammatory effects. (abst - 2012) http/www.ncbi.nlm.nih.gov/pubmed/22342378
Cannabinoid receptor 2 agonists inhibit migration of activated dendritic cells via modulation of MMP-9 (abst – 2012) http/www.jimmunol.org/cgi/content/meeting_abstract/188/1_MeetingAbstracts/173.23?maxtoshow=&hits=25&RESULTFORMAT=&fulltext=cannabinoid&searchid=1&FIRSTINDEX=130&sortspec=date&resourcetype=HWCIT
Genetic Background Can Result in a Marked or Minimal Effect of Gene Knockout (GPR55 and CB2 Receptor) in Experimental Autoimmune Encephalomyelitis Models of Multiple Sclerosis. (full – 2013) http/www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0076907
Multiple sclerosis and the blood-central nervous system barrier. (full – 2013) http/www.ncbi.nlm.nih.gov/pmc/articles/PMC3562587/
Cannabidiol provides long-lasting protection against the deleterious effects of inflammation in a viral model of multiple sclerosis: A role for A2A receptors. (full – 2013) http/www.sciencedirect.com/science/article/pii/S0969996113001939
Selective CB2 receptor activation ameliorates EAE by reducing Th17 differentiation and immune cell accumulation in the CNS. (abst – 2013) http/www.ncbi.nlm.nih.gov/pubmed/24342422
Pre- and postsynaptic type-1 cannabinoid receptors control the alterations of glutamate transmission in experimental autoimmune encephalomyelitis. (abst – 2014) http/www.ncbi.nlm.nih.gov/pubmed/24440366
Activating Cannabinoid Receptor 2 Alleviates Pathogenesis of Experimental Autoimmune Encephalomyelitis Via Activation of Autophagy and Inhibiting NLRP3 Inflammasome. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/25417929
Neuroprotection in Experimental Autoimmune Encephalomyelitis and Progressive Multiple Sclerosis by Cannabis-Based Cannabinoids. (abst – 2014) http://www.ncbi.nlm.nih.gov/pubmed/25537576
Interaction between the protective effects of cannabidiol and palmitoylethanolamide in experimental model of multiple sclerosis in C57BL/6 mice (abst – 2015) http://www.sciencedirect.com/science...06452215000858